ASSESSING THE IMPACT ON PEFR AMONG SMOKERS AND PASSIVE SMOKERS–A COMPARATIVE STUDY

Authors

  • GANESH PANDIAN B. Unit of Clinical Pharmacy and Pharmacy Practice, Faculty of Pharmacy, AIMST University, Malaysia, Department of Pharmacy Practice, Shri Vishnu College of Pharmacy, Andhrapradesh, India
  • SIREESHA P. Unit of Clinical Pharmacy and Pharmacy Practice, Faculty of Pharmacy, AIMST University, Malaysia https://orcid.org/0000-0002-2558-8280
  • YEN PING N. G. Unit of Clinical Pharmacy and Pharmacy Practice, Faculty of Pharmacy, AIMST University, Malaysia https://orcid.org/0000-0002-5272-2588
  • SUSMITHA G. Department of Pharmacy Practice, Shri Vishnu College of Pharmacy, Andhrapradesh, India, Bhaskar College of Pharmacy, Moinabad, India
  • SAM AASEER T. Unit of Clinical Pharmacy and Pharmacy Practice, Faculty of Pharmacy, AIMST University, Malaysia

DOI:

https://doi.org/10.22159/ijpps.2019v11i11.35058

Keywords:

Respiratory Function Tests, Peak Expiratory Flow Rate, Smoking

Abstract

Objective: The purpose of this study was to monitor the intensity and difference in Peak Expiratory Flow Rate (PEFR) between smokers and passive smokers.

Methods: A total of 1000 participants were enrolled in two groups as smokers and passive smokers who are living closely with smokers. Their PEFR values were measured with Wright’s mini peak flow meter. The influence of smoking on the lung function among smokers and passive smokers were assessed with a suitable statistical test.

Results: Among the study participants, most of the smokers were in the age group of 31 to 60 and 31 to 50 in passive smokers. Based on the lung function smokers (31%) and passive smokers (19.2%) were in the red zone, PEFR was decreased in both smokers as well as passive smokers, and the magnitude of decline was higher in passive smoking elderly individuals. The impact of passive smoking was significantly observed in all the categories of smoking history they are living with.

Conclusion: Smokers and passive smokers have equally deleterious effects on PEFR. Where passive smoking emerged as the main variable to influence airway obstruction in smokers that caused a greater reduction in PEFR.

Downloads

Download data is not yet available.

References

World Health Organization (WHO) report on the Global Tobacco Epidemic; 2017. Available from: http://www.who.int/ tobacco/global_report/en/. [Last accessed on 20 May 2018]

Avsar A, Darka O, Topaloglu B, Bek Y. Association of passive smoking with carries and related salivary biomarkers in young children. Arch Oral Biol 2008;53:969-74.

Oberg M, Peruga A, Pruss Ustun A, Sjaakkola M, Woodward A. Worldwide burden of disease from exposure to second-hand smoke: a retrospective analysis of data from 192 countries. Lancet 2011;377:139-46.

Office on Smoking and Health (US). The Health Consequences of Involuntary Exposure to Tobacco Smoke: A Report of the Surgeon General. Atlanta (GA): Centers for Disease Control and Prevention (US); 2006. Available from: https://www.ncbi. nlm.nih.gov/books/NBK44324/. [Last accessed on 10 Mar 2018]

US EPA. Respiratory health effects of passive smoking: lung cancer and other disorders. U. S. Environmental Protection Agency, Office of Research and Development, Office of Health and Environmental Assessment, Washington, DC, EPA/600/6-90/006F; 1992.

Jansson C, Chinn S, Jarvis D, Zock JP, Toren K, Burney P. Effects of passive smoking on respiratory symptoms, bronchial responsiveness, lung function, and total IgE in the European Community respiratory health survey: a cross-sectional study. Lancet 2001;358:2103–9.

What's new in nicotine and tobacco research? Nicotine Tobacco Research 2003;5:281-7. Available from: https://doi.org/ 10.1080/1462220031000095717 [Last accessed on 10 Jul 2019].

Nida BSA, Natasha H, Nahlah EI. Smoking status affecting survival of adenocarcinoma lung cancer patients in Kuala Lumpur, Malaysia. Asian J Pharm Clin Res 2017;10:312-3.

Mathers CD, Loncar D. Projections of global mortality and Barden of disease from 2002 to 2030. Plos Med 2006;3:e442.

World Health Organization, CDC. Global Adult Tobacco Survey 2 (GATS 2): India factsheet; 2016-2017. Available from: http://www.who.int/tobacco/surveillance/survey/gats/GATS_India_2016-17_FactSheet. pdf?ua=1. [Last accessed on 10 Jun 2018]

Tambi M, Rao BN, Glad MMI, Praveen KM. Effect of cigar and cigarette smoking on peak expiratory flow rate. J Clin Diagnostic Res 2013;7:1886-9.

Wright BM, McKerrow CB. Maximum forced expiratory flow rate as a measure of ventilatory capacity: with a description of a new portable instrument for measuring it. Br Med J 1959;2:1041-6.

Omair A, Kazmi T, Alam S. Smoking prevalence and awareness about tobacco related diseases among medical students of Ziauddin Medical University. J Pak Med Assoc 2002;52:389-92.

Jain SK, Rajendra K, Sharma DA. Factors influencing peak expiratory flow rate in normal subjects-II. Lung India 1983;1:92-7.

Forey BA, Thornton AJ, Lee PN. Systematic review with meta-analysis of the epidemiological evidence relating smoking to COPD, chronic bronchitis and emphysema. BMC Pulm Med 2011;11:36.

Published

01-11-2019

How to Cite

B., G. P., S. P., Y. P. N. G., S. G., and S. A. T. “ASSESSING THE IMPACT ON PEFR AMONG SMOKERS AND PASSIVE SMOKERS–A COMPARATIVE STUDY”. International Journal of Pharmacy and Pharmaceutical Sciences, vol. 11, no. 11, Nov. 2019, pp. 17-19, doi:10.22159/ijpps.2019v11i11.35058.

Issue

Section

Original Article(s)